Targeting Inflammation in Atherosclerosis

Targeting inflammation in atherosclerosis is an emerging strategy to treat cardiovascular diseases. Atherosclerosis, the buildup of fatty plaques in arteries, is driven not only by cholesterol but also by chronic inflammation. Inflammatory cells, such as macrophages, infiltrate plaque sites and exacerbate plaque formation, making it unstable and prone to rupture, which can lead to heart attacks or strokes.

Recent research suggests that reducing inflammation could slow or even reverse atherosclerosis. Anti-inflammatory treatments aim to target key inflammatory pathways involved in plaque development. For instance, drugs like canakinumab, which inhibit the interleukin-1β (IL-1β) pathway, have shown promise in reducing inflammation and lowering cardiovascular events. Additionally, statins, commonly used to lower cholesterol, have anti-inflammatory effects that contribute to their cardiovascular benefits.

By focusing on inflammation, researchers are developing new therapies that may complement traditional treatments, providing a more comprehensive approach to preventing and managing atherosclerosis and its associated risks.

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